Can you reduce your risk of cancer by 50%?

March 2014
 by Gabriel MacSharry
In some respects cancer is to industrialized countries today what tuberculosis was to the 18th and 19th century: a major cause of death and misery which defeats the best efforts of conventional medicine.

Please check out this graph that illustrates the relationship between latitude, sun exposure and death rates from cancer.

Graph Explained

The vertical axis shows the number of cancer-related deaths per 100,000, while the horizontal axis indicates latitude. It clearly shows that cancer-deaths go up significantly, the farther away from the equator one lives.

With sun exposure, UVB radiation from the sun converts cholesterol to vitamin D, one of the most potent anticancer vitamins. Your vitamin D levels should be kept in a healthy range. If you're taking high amounts of oral vitamin D in a supplement, you need to measure your vitamin D blood levels to avoid overdosing. Talk to your GP regarding this test. The correct test to ask for is 25(OH)D, also called 25-hydroxyvitamin D.

So where does one get vitamin D?

Vitamin D From Food

What the research on vitamin D tells us is that unless you are a fisherman, farmer, or otherwise outdoors and exposed regularly to sunlight, living in your ancestral latitude (more on this later), you are unlikely to obtain adequate amounts of vitamin D from the sun. Historically the balance of one's daily need was provided by food. Primitive peoples instinctively chose vitamin-D-rich foods including the intestines, organ meats, skin and fat from certain land animals, as well as shellfish, oily fish and insects. Many of these foods are unacceptable to the modern palate.

For food sources to provide us with D the source must be sunlight exposed. With exposure to UV-B sunlight, vitamin D is produced from fat in the fur, feathers, and skin of animals, birds and reptiles. Carnivores get additional D from the tissues and organs of their prey. Lichen contains vitamin D and may provide a source of vitamin D in the UV-B sunlight-poor northern latitudes.1 Vitamin D content will vary in the organs and tissues of animals, pigs, cows, and sheep, depending on the amount of time spent in UV-B containing sunlight and/or how much D is given as a supplement. Poultry and eggs contain varying amounts of vitamin D obtained from insects, fishmeal, and sunlight containing UV-B or supplements

Fish, unlike mammals, birds and reptiles, do not respond to sunlight and rely on vitamin D found in phytoplankton and other fish. Salmon must feed on phytoplankton and fish in order to obtain and store significant vitamin D in their fat, flesh, skin, and organs. Thus, modern farm-raised salmon, unless artificially supplemented, may be a poor source of this essential nutrient.

Vitamin D From Sunlight

Pick up any popular book on vitamins and you will read that ten minutes of daily exposure of the arms and legs to sunlight will supply us with all the vitamin D that we need. Humans do indeed manufacture vitamin D from cholesterol by the action of sunlight on the skin but it is actually very difficult to obtain even a minimal amount of vitamin D with a brief foray into the sunlight.2,3

Ultraviolet (UV) light is divided into 3 bands or wavelength ranges, which are referred to as UV-C, UV-B and UV-A.4 UV-C is the most energetic and shortest of the UV bands. It will burn human skin rapidly in extremely small doses. Fortunately, it is completely absorbed by the ozone layer. However, UV-C is present in some lights. For this reason, fluorescent and halogen and other specialty lights may contribute to skin cancer.

UV-A, known as the "tanning ray," is primarily responsible for darkening the pigment in our skin. Most tanning bulbs have a high UV-A output, with a small percentage of UV-B. UV-A is less energetic than UV-B, so exposure to UV-A will not result in a burn, unless the skin is photosensitive or excessive doses are used. UV-A penetrates more deeply into the skin than UV-B, due to its longer wavelength. Until recently, UV-A was not blocked by sunscreens. It is now considered to be a major contributor to the high incidence of non-melanoma skin cancers.5 Seventy-eight percent of UV-A penetrates glass so windows do not offer protection.

The ultraviolet wavelength that stimulates our bodies to produce vitamin D is UV-B. It is sometimes called the "burning ray" because it is the primary cause of sunburn (erythema). However, UV-B initiates beneficial responses, stimulating the production of vitamin D that the body uses in many important processes. Although UV-B causes sunburn, it also causes special skin cells called melanocytes to produce melanin, which is protective. UV-B also stimulates the production of Melanocyte Stimulating Hormone (MSH), an important hormone in weight loss and energy production.6

The reason it is difficult to get adequate vitamin D from sunlight is that while UV-A is present throughout the day, the amount of UV-B present has to do with the angle of the sun's rays. Thus, UV-B is present only during midday hours at higher latitudes, and only with significant intensity in temperate or tropical latitudes. Only 5 percent of the UV-B light range goes through glass and it does not penetrate clouds, smog or fog.

Sun exposure at higher latitudes before 10 am or after 2 pm will cause burning from UV-A before it will supply adequate vitamin D from UV-B. This finding may surprise you, as it did the researchers. It means that sunning must occur between the hours we have been told to avoid. Only sunning between 10 am and 2 pm during summer months (or winter months in southern latitudes) for 20-120 minutes, depending on skin type and color, will form adequate vitamin D before burning occurs.7

It takes about 24 hours for UV-B-stimulated vitamin D to show up as maximum levels of vitamin D in the blood. Cholesterol-containing body oils are critical to this absorption process.8 Because the body needs 30-60 minutes to absorb these vitamin-D-containing oils, it is best to delay showering or bathing for one hour after exposure. The skin oils in which vitamin D is produced can also be removed by chlorine in swimming pools.

The current suggested exposure of hands, face and arms for 10-20 minutes, three times a week, provides only 200-400 IU of vitamin D each time or an average of 100-200 IU per day during the summer months. In order to achieve optimal levels of vitamin D, 85 percent of body surface needs exposure to prime midday sun. (About 100-200 IU of vitamin D is produced for each 5 percent of body surface exposed, we want 4,000 iu.) Light skinned people need 10-20 minutes of exposure while dark skinned people need 90-120 minutes.9

Latitude and altitude determine the intensity of UV light. UV-B is stronger at higher altitudes. Latitudes higher than 30° (both north and south) have insufficient UV-B sunlight two to six months of the year, even at midday.10 Latitudes higher than 40° have insufficient sunlight to achieve optimum levels of D during six to eight months of the year. In much of the US, which is between 30° and 45° latitude, six months or more during each year have insufficient UV-B sunlight to produce optimal D levels. In far northern or southern locations, latitudes 45° and higher, even summer sun is too weak to provide optimum levels of vitamin D.11-13 A simple meter is available to determine UV-B levels where you live.

Sunlight on the Inside

If any nutrient incorporates the properties of sunlight, it is vitamin D. The healthy "primitive" peoples that Dr. Price observed not only had broad, round, "sunny" faces, they also had sunny dispositions and optimistic attitudes towards life in spite of many hardships. Typical food intakes for peoples who have not been "civilized" range from 3,000 IU-6,000 IU. Modern intakes are paltry in comparison. The standard American diet provides vitamin D only in very low quantities.

The first step towards redressing some of the ills of civilized life—from depression to road rage, from cavities to osteoporosis—would be to get more light, inside or outside. Vitamin D adds sunlight to life from childhood through the golden years. In nonagenarians and centagenarians high levels of vitamin D in the blood and normal thyroid function were the strongest markers of health and longevity.14

Whether in the form of sunlight or dietary vitamin D from food and fish oils, optimal levels of the sunshine vitamin allow your body and mind to thrive, even during periods of stress.

The bottom line for people living in Ireland is to maximise the amount of sun exposure we get (try to get at least 20 mins of sun exposure to your face and arms daily), consume oily fish on aregular basis or supplement with a good quality cod liver oil, consume organic eggs, cheese and butter and make a healthy bone broth soup once a week.


  1. Bjorn LO, Wang T. Vitamin D in an ecological context. Int.J.Circumpolar.Health 2000;59:26-32.
  2. Glerup H, Mikkelsen K, Poulsen L et al. Commonly recommended daily intake of vitamin D is not sufficient if sunlight exposure is limited. J.Intern.Med. 2000;247:260-8.
  3. Glerup H, Eriksen EF. [Vitamin D deficiency. Easy to diagnose, often overlooked (see comments)]. Ugeskr.Laeger 1999;161:2515-21. 6. Diffey BL. Solar ultraviolet radiation effects on biological systems. Phys.Med.Biol. 1991;36:299-328.
  4. Ibid
  5. Moan J, Dahlback A, Setlow RB. Epidemiological support for an hypothesis for melanoma induction indicating a role for UVA radiation. Photochem.Photobiol. 1999;70:243-7.
  6. Ranson M, Posen S, Mason RS. Human melanocytes as a target tissue for hormones: in vitro studies with 1 alpha-25, dihydroxyvitamin D3, alpha-melanocyte stimulating hormone, and beta-estradiol. J.Invest Dermatol.1988;91:593-8.
  7. Sayre, R. M., Dowdy, J. C., Shepherd, J., Sadig, I., Bager, A., and Kollias, N. Vitamin D Production by Natural and Artificial Sources. 1998. Orlando, Florida, Photo Medical Society Meeting. 3-1-1998. Ref Type: Conference Proceeding
  8. Holick MF. The cutaneous photosynthesis of previtamin D3: a unique photoendocrine system. J.Invest Dermatol. 1981;77:51-8.
  9. Matsuoka LY, Wortsman J, Haddad JG, Kolm P, Hollis BW. Racial pigmentation and the cutaneous synthesis of vitamin D [see comments]. Arch.Dermatol. 1991;127:536-8.
  10. Matsuoka LY, Wortsman J, Haddad JG, Hollis BW. In vivo threshold for cutaneous synthesis of vitamin D3. J.Lab Clin.Med. 1989;114:301-5.
  11. Season, latitude, and ability of sunlight to promote synthesis of vitamin D3 in skin. Nutr.Rev. 1989;47:252-3.
  12. Pettifor JM, Moodley GP, Hough FS et al. The effect of season and latitude on in vitro vitamin D formation by sunlight in South Africa. S.Afr.Med.J. 1996;86:1270-2.
  13. Webb AR, Kline L, Holick MF. Influence of season and latitude on the cutaneous synthesis of vitamin D3: exposure to winter sunlight in Boston and Edmonton will not promote vitamin D3 synthesis in human skin. J.Clin.Endocrinol.Metab 1988;67:373-8.
  14. Mariani E, Ravaglia G, Forti P et al. Vitamin D, thyroid hormones and muscle mass influence natural killer (NK) innate immunity in healthy nonagenarians and centenarians [published erratum appears in Clin Exp Immunol 1999 Jul;117(1):206]. Clin.Exp.Immunol.

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